Discovering Hope: Longeveron’s Groundbreaking Lomecel-B Data for Alzheimer’s Disease Unveiled at CTAD24 Conference

Discovering Hope: Longeveron’s Groundbreaking Lomecel-B Data for Alzheimer’s Disease Unveiled at CTAD24 Conference

Findings offer potential mechanistic and clinical insights in the development of cellular therapy Lomecel-B™ for mild Alzheimer’s disease (AD)

Lomecel-B™ capacity to inhibit MMP14 correlates with improved clinical and biomarker outcomes in mild AD. The immunomodulatory and pro-vascular effects of Lomecel-B™ in mild AD are potentially driven by its ability to exert MMP14 inhibition, which may protect TIE2 receptor integrity in human AD patients. MIAMI, Oct. 29, 2024 (GLOBE NEWSWIRE) — Longeveron Inc. (NASDAQ: LGVN), a clinical stage regenerative medicine biotechnology company developing cellular therapies for life-threatening and chronic aging-related conditions, today announced that its submission entitled “Lomecel-B inhibition of MMP14 activity predicts Lomecel-B bioactivity in the treatment of mild Alzheimer’s disease” was presented as a late breaking poster presentation at the 17th edition of the Clinical Trials on Alzheimer’s Disease Conference (CTAD24) taking place October 29 -November 1, 2024 in Madrid, Spain. “As a medicinal signaling cell therapy that has multiple potential mechanisms of action to address inflammatory responses in the brain, Lomecel-B™ offers the potential to address the underlying pathology of Alzheimer’s disease,” said Joshua Hare, M.D.

How this will affect you:

As research into Lomecel-B™ progresses, there is hope for a potential breakthrough in the treatment of mild Alzheimer’s disease. If successful, this cellular therapy could offer new possibilities for those suffering from this debilitating condition, providing improved clinical outcomes and potentially slowing down the progression of the disease.

How this will affect the world:

The unveiling of Longeveron’s groundbreaking data at the CTAD24 Conference marks a significant step forward in the field of Alzheimer’s research. The potential mechanistic and clinical insights gained from this study could have far-reaching implications for the development of future treatments for AD, ultimately improving the quality of life for millions of individuals worldwide.

Conclusion:

The presentation of Longeveron’s findings at the CTAD24 Conference brings a renewed sense of hope in the fight against Alzheimer’s disease. With the promising results of Lomecel-B™ inhibition of MMP14 activity, there is optimism for the development of effective cellular therapies that could change the landscape of Alzheimer’s treatment and offer new possibilities for patients and their loved ones.

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